Interesting. Oxalates do explain different locations. But is there a mechanism why symptoms would appear in the legs (as they do for most mild cases)? Maybe for the same reason that gout often is connected to inflamed toes, for blood circulation reasons (I assume) that cause gout crystals to form there? Food for thought.XenMan wrote: ↑Mon Sep 28, 2020 9:16 amAs my RLS is from oxalates, symptoms occur when oxalates are broken down though exercise or general oxalate dumping. I have nerve and the corresponding muscle trigger points. It seems the oxalate leaves the nerves and makes them ‘unhappy’ and they activate the corresponding muscles, which then create the motor sensory discrepancy for symptoms to exist. I can press the nerves to stop symptoms or treat the muscles.
My symptoms move all over place with oxalate dumping with some easy, and others very difficult to treat.
My symptoms have always been symmetrical, either both legs, or both arms, or whole body. I never really thought about this before. Maybe I assumed that my legs are under more stress/pressure or whatever, as symptoms were confined to my legs until I augmented. But this clearly doesn't explain why I would get "restless arms" (with no symptoms in my legs) when I tried Gabapentin. But always both arms, too.
My dream was always to have a huge RLS database, and do a statistical analysis on that. Enter what treatments helps (for example anything with pressure/massages or heat), which supplements did or did not help, which drugs are effective, sensations (painful RLS or "electric" RLS)... I wouldn't be surprised if we would find different stereotypes of RLS, that may be due to different underlying root causes. Could it be that people with asymmetrical symptoms (only one leg at a time, for example) have different root causes than people with symmetrical locations (where it could be more likely that the issue is in the brain and not in the extremities)?